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Representative fundus photographs (A–D) and FFA (E–H) of the retinas in the NC and EIU groups. (A) Fundus photograph from the WT NC group. (B) Fundus photograph from the KO NC group. (C) Fundus photograph from the WT EIU group. (D) Fundus photograph from the KO EIU group. (E) FFA from the WT NC group. (F) FFA from the KO NC group. (G) FFA from the WT EIU group. (H) FFA from the KO EIU group. (I) Grade of the severity of vitreous opacity. ∗∗ P < 0.01. n = 3 biological replicates per group. FFA, fundus <t>fluorescein</t> angiography; WT, wild type; KO, Cgas knockout; NC, normal control; EIU, endotoxin-induced uveitis.
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Representative fundus photographs (A–D) and FFA (E–H) of the retinas in the NC and EIU groups. (A) Fundus photograph from the WT NC group. (B) Fundus photograph from the KO NC group. (C) Fundus photograph from the WT EIU group. (D) Fundus photograph from the KO EIU group. (E) FFA from the WT NC group. (F) FFA from the KO NC group. (G) FFA from the WT EIU group. (H) FFA from the KO EIU group. (I) Grade of the severity of vitreous opacity. ∗∗ P < 0.01. n = 3 biological replicates per group. FFA, fundus <t>fluorescein</t> angiography; WT, wild type; KO, Cgas knockout; NC, normal control; EIU, endotoxin-induced uveitis.
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Representative fundus photographs (A–D) and FFA (E–H) of the retinas in the NC and EIU groups. (A) Fundus photograph from the WT NC group. (B) Fundus photograph from the KO NC group. (C) Fundus photograph from the WT EIU group. (D) Fundus photograph from the KO EIU group. (E) FFA from the WT NC group. (F) FFA from the KO NC group. (G) FFA from the WT EIU group. (H) FFA from the KO EIU group. (I) Grade of the severity of vitreous opacity. ∗∗ P < 0.01. n = 3 biological replicates per group. FFA, fundus <t>fluorescein</t> angiography; WT, wild type; KO, Cgas knockout; NC, normal control; EIU, endotoxin-induced uveitis.
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Representative fundus photographs (A–D) and FFA (E–H) of the retinas in the NC and EIU groups. (A) Fundus photograph from the WT NC group. (B) Fundus photograph from the KO NC group. (C) Fundus photograph from the WT EIU group. (D) Fundus photograph from the KO EIU group. (E) FFA from the WT NC group. (F) FFA from the KO NC group. (G) FFA from the WT EIU group. (H) FFA from the KO EIU group. (I) Grade of the severity of vitreous opacity. ∗∗ P < 0.01. n = 3 biological replicates per group. FFA, fundus <t>fluorescein</t> angiography; WT, wild type; KO, Cgas knockout; NC, normal control; EIU, endotoxin-induced uveitis.
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Representative fundus photographs (A–D) and FFA (E–H) of the retinas in the NC and EIU groups. (A) Fundus photograph from the WT NC group. (B) Fundus photograph from the KO NC group. (C) Fundus photograph from the WT EIU group. (D) Fundus photograph from the KO EIU group. (E) FFA from the WT NC group. (F) FFA from the KO NC group. (G) FFA from the WT EIU group. (H) FFA from the KO EIU group. (I) Grade of the severity of vitreous opacity. ∗∗ P < 0.01. n = 3 biological replicates per group. FFA, fundus <t>fluorescein</t> angiography; WT, wild type; KO, Cgas knockout; NC, normal control; EIU, endotoxin-induced uveitis.
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Representative fundus photographs (A–D) and FFA (E–H) of the retinas in the NC and EIU groups. (A) Fundus photograph from the WT NC group. (B) Fundus photograph from the KO NC group. (C) Fundus photograph from the WT EIU group. (D) Fundus photograph from the KO EIU group. (E) FFA from the WT NC group. (F) FFA from the KO NC group. (G) FFA from the WT EIU group. (H) FFA from the KO EIU group. (I) Grade of the severity of vitreous opacity. ∗∗ P < 0.01. n = 3 biological replicates per group. FFA, fundus <t>fluorescein</t> angiography; WT, wild type; KO, Cgas knockout; NC, normal control; EIU, endotoxin-induced uveitis.
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Growth inhibition of HT-29 (panel A ), A549 (panel B ), MCF-7 (panel C ), HeLa (panel D and <t>CCD</t> <t>841</t> CON (panel E ) cells treated with AE extracts for different exposure times assessed using MTT assay. The grey broken lines in the graphs mark the level of IC10 and IC 50 values corresponding to 90% and 50% cell growth, respectively, compared to control (100%). The heatmap of IC 50 values on AE extract on different cell lines (panel G ). The genotoxic effects of AE on HT-29 cells expressed as % DNA in the comet tail (panel F ). Results represent means ± SD from three independent experiments carried out in triplicates. The p-value for 0.5 mg DW/mL concentration was 0.0060
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Image Search Results


Representative fundus photographs (A–D) and FFA (E–H) of the retinas in the NC and EIU groups. (A) Fundus photograph from the WT NC group. (B) Fundus photograph from the KO NC group. (C) Fundus photograph from the WT EIU group. (D) Fundus photograph from the KO EIU group. (E) FFA from the WT NC group. (F) FFA from the KO NC group. (G) FFA from the WT EIU group. (H) FFA from the KO EIU group. (I) Grade of the severity of vitreous opacity. ∗∗ P < 0.01. n = 3 biological replicates per group. FFA, fundus fluorescein angiography; WT, wild type; KO, Cgas knockout; NC, normal control; EIU, endotoxin-induced uveitis.

Journal: Genes & Diseases

Article Title: cGAS knockout inhibited endotoxin-induced uveitis in mice

doi: 10.1016/j.gendis.2025.101786

Figure Lengend Snippet: Representative fundus photographs (A–D) and FFA (E–H) of the retinas in the NC and EIU groups. (A) Fundus photograph from the WT NC group. (B) Fundus photograph from the KO NC group. (C) Fundus photograph from the WT EIU group. (D) Fundus photograph from the KO EIU group. (E) FFA from the WT NC group. (F) FFA from the KO NC group. (G) FFA from the WT EIU group. (H) FFA from the KO EIU group. (I) Grade of the severity of vitreous opacity. ∗∗ P < 0.01. n = 3 biological replicates per group. FFA, fundus fluorescein angiography; WT, wild type; KO, Cgas knockout; NC, normal control; EIU, endotoxin-induced uveitis.

Article Snippet: Concanavalin A and fluorescein (FL-1001-25; Vectorlabs) were dissolved in PBS.

Techniques: Knock-Out, Control

Growth inhibition of HT-29 (panel A ), A549 (panel B ), MCF-7 (panel C ), HeLa (panel D and CCD 841 CON (panel E ) cells treated with AE extracts for different exposure times assessed using MTT assay. The grey broken lines in the graphs mark the level of IC10 and IC 50 values corresponding to 90% and 50% cell growth, respectively, compared to control (100%). The heatmap of IC 50 values on AE extract on different cell lines (panel G ). The genotoxic effects of AE on HT-29 cells expressed as % DNA in the comet tail (panel F ). Results represent means ± SD from three independent experiments carried out in triplicates. The p-value for 0.5 mg DW/mL concentration was 0.0060

Journal: BMC Complementary Medicine and Therapies

Article Title: Inhibition of the PI3K signaling pathway in cancer cells by Agrimonia eupatoria L. ethanolic extract: identification of tricoumaroyl spermidine as a potential PI3K inhibitor

doi: 10.1186/s12906-025-05231-z

Figure Lengend Snippet: Growth inhibition of HT-29 (panel A ), A549 (panel B ), MCF-7 (panel C ), HeLa (panel D and CCD 841 CON (panel E ) cells treated with AE extracts for different exposure times assessed using MTT assay. The grey broken lines in the graphs mark the level of IC10 and IC 50 values corresponding to 90% and 50% cell growth, respectively, compared to control (100%). The heatmap of IC 50 values on AE extract on different cell lines (panel G ). The genotoxic effects of AE on HT-29 cells expressed as % DNA in the comet tail (panel F ). Results represent means ± SD from three independent experiments carried out in triplicates. The p-value for 0.5 mg DW/mL concentration was 0.0060

Article Snippet: MCF-7 (ATCC HTB‐22) (human breast cancer), HT-29 (ATCC HTB‐38) (Human colon adenocarcinoma), A549 (ATCC CCL-185) (human lung adenocarcinoma) and HeLa (ATCC CCL-2) (human cervical carcinoma) CCD 841 CoN (ATCC CRL-1790) (non-cancerous human colon fibroblast cells) cell cultures were obtained from ATCC.

Techniques: Inhibition, MTT Assay, Control, Concentration Assay